Neuropathy due to lead poisoning

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Cerebellar ataxia and peripheral neuropathy due to chronic bromvalerylurea poisoning.

A patient with chronic bromvalerylurea poisoning showed cerebellar ataxia and peripheral neuropathy. The patient was a 42-year-old Japanese man who developed consciousness disturbance, diplopia, slurred speech, ataxia and gait disturbance after having taken bromvalerylurea for ten years. Magnetic resonance imaging revealed atrophy of the cerebellum and pontine tegmentum. An electrophysiological...

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Anaemia and abdominal pain due to occupational lead poisoning.

We describe a 47-year-old patient with chronic anaemia with basophilic stippling of erythrocytes, recurrent abdominal colics, discoloration of gums, sensitive polyneuropathy to the four limbs, hyperuricaemia, hepatosteatosis with raised transaminases, and a long ignored history of lead exposure in a battery recycling plant. The diagnosis of poisoning was confirmed by high lead levels in the blo...

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Lead poisoning due to geophagia: The consumption of miniature pottery

Geophagia (the pica of pottery, clay, earth, or dirt) is practiced before and during pregnancy in several countries, including Mexico, Turkey, Australia, and some African countries, and has been linked with cultural fertility beliefs and the satisfaction of cravings. Unfortunately, consumption of contaminated pottery can represent a source of lead exposure. Concerns regarding ingested pottery a...

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Appendectomy due to lead poisoning: a case-report

BACKGROUND Lead poisoning is a common occupational health hazard in developing countries and many misdiagnoses and malpractices may occur due to unawareness of lead poisoning symptoms. CASE PRESENTATION We report a case of occupational lead poisoning in an adult battery worker with abdominal colic who initially underwent appendectomy with removal of normal appendix. Later on he was diagnosed ...

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A case of lead poisoning due to snooker chalk.

A 3 year, 9 month old child with pica presented with a blood lead concentration of 1.74 micromol/l (360 microg/l). The source of poisoning was snooker chalk (lead content 7200 microg/g). She was treated with intravenous calcium disodium edetate chelation. Thirty months later her blood lead was 0.39 micromol/l (80 microg/l). This case illustrates the need to be vigilant for more unusual causes o...

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ژورنال

عنوان ژورنال: Annals of Indian Academy of Neurology

سال: 2010

ISSN: 0972-2327

DOI: 10.4103/0972-2327.61287